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Unlike symptomatic treatments (gabapentinoids, duloxetine), ALC promotes actual nerve fiber regeneration and addresses underlying pathophysiology.[17][2] This disease-modifying potential makes it particularly valuable for early intervention
Unlike injectable formulations that typically require cold-chain storage, oral GLP-1RAs bypass refrigeration constraints, thereby reducing manufacturing and distribution costs and improving accessibility in resource-limited settings [73]
2004;109:962-s65
Endogenous LAC levels range between 8 and 9 g/g wet rodent tissue corresponding to 4045 nmol/g wet tissue as previously reported in the rodent brain [30, 31]
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